Mandibular cementoblastoma: Case report (2024)

Open Journal of Stomatology
Vol.2No.1(2012), Article ID:18234,4 pages DOI:10.4236/ojst.2012.21009

Mandibular cementoblastoma: Case report

Cintia Mussi Milani1*, César Augusto Thomé2, Rosangela Sayuri Saga Kamikawa3, Mariana Daniel da Silva4, Maria Angela Naval Machado5

Mandibular cementoblastoma: Case report (1)

1Universidade Tuiuti do Paraná, Curitiba, Brazil

2Center for Health and Biological Sciences, Pontifícia Universidade Católica do Paraná, Curitiba, Brazil

3Associação Brasileira de Odontologia-Seção Paraná, Curitiba, Brazil

4Private Practice, Curitiba, Brazil

5Universidade Federal do Paraná, Curitiba, Brazil

Email: *cintiammilani@hotmail.com

Received 28 November 2011; revised 12 January 2012; accepted 9 February 2012

Keywords: Odontogenic Tumor; Cementoblasts; Oral Surgery

ABSTRACT

Cementoblastoma is a rare benign lesion that represents less than 1% of all odontogenic tumours. It’s characterized by proliferation of cementum-like tissue and in almost all cases tends to be associated with an erupted permanent tooth, most often the first molar. We present an unusual case of a large cementoblastoma that affected the right mandibular body, extending from the first premolar to the second molar, of a 19-years-old male. In this case an initial surgery was attempted under local anaesthesia, resulting in incomplete tumor removal. A second surgical procedure was performed under general anaesthesia, ensuring the complete excision of the lesion. The patient was monitored for 1 year after surgery and did not show any signs of recurrence.

1. INTRODUCTION

Cementoblastoma is currently ranked by the World Health Organization as an odontogenic mesenchyme or ectomesenchyme tumor, with or without involvement of the odontogenic epithelium [1]. It is a benign tumor that is characterized by proliferation cementum-like tissue that is attached to the roots of teeth [2,3-6].

A cementoblastoma lesion is usually found in the mandible, most commonly in the pre-molar and molar region [7,8]. Its prevalence is higher in young male adults who are under 30 years of age [9]. Cementoblastomas are slow growing lesions [8-10] and are usually asymptomatic; however pain, and swelling may occur [2,3,7-15].

Radiographically, the lesion presents as a radiopaque mass that is attached to the root tooth, which is usually vital and is surrounded by a radiolucent halo [1,8-10,13, 16]. Histologically, cementoblastoma tissue is similar to cementum.

The clinical and radiological findings are of great importance for the differential diagnosis of odontogenic neoplasms [8], which must include osteoblastoma, osteosarcoma and focal sclerosing osteomyelitis [8,15].

The recommended treatment consists of the surgical removal of the tumor and associated teeth [2]. In cases in which an early diagnosis is made, the lesion can be removed in association with the endodontic treatment of the involved tooth [17]. If the lesion is not completely removed [2,10], recurrence and continued growth can be observed due to its unlimited growth potential [2,8,17], but this is not common [2,8,10].

We present an unusual case of a residual cementoblastoma involving the right mandibular body of a 19- years-old male.

2. CASE REPORT

A 19-year-old, healthy Brazilian man, was referred to a maxillofacial surgeon due to the presence of a cementoblastoma in the right mandibular body. The patient reported that he was being treated at another clinic, and his initial complaint was related to a progressive facial asymmetry.

According to the patient, the surgeon who initially treated him requested a panoramic radiograph (Figure 1) and a cone-beam tomography (Figure 2) and performed a biopsy, that led to the diagnosis of cementoblastoma. Surgery to remove the entire lesion was performed under local anesthesia, resulting in severe intraoperative pain and incomplete removal of the lesion. This initial surgery was performed three months prior to his presentation.

On clinical examination, the patient presented a painless hard swelling in the right lower posterior buccal and lingual gingiva, extending from the region of the

Mandibular cementoblastoma: Case report (2)

Figure 1. Panoramic radiograph showing a radiopaque lesion that is surrounded by a radiolucent halo and is in contact with the roots of the mandibular second premolar and the first and second molars.

Mandibular cementoblastoma: Case report (3)

Figure 2. Cone-beam tomography showing the buccal and lingual extension of the lesion.

second premolar to the second molar on the right. These teeth were removed during the first surgery.

The initial panoramic radiograph revealed a well-defined, round, radiopaque lesion, that was delineated by a thin radiolucent halo, and was in contact with the roots of the second premolar, and the first and second right lower molars. The radiographic appearance was pathognomonic of cementoblastoma. Tomography showed the buccal and lingual extension of the lesion. The panoramic radiograph performed after the first surgery revealed that a large amount of the lesion was left in place, and a fracture in the apical portion of the mesial root of the third molar occurred intraoperatively (Figure 3). A vitality test of the third molar revealed that this tooth was still vital.

Based on clinical and radiological findings, surgery under general anesthesia was planned to remove the remainder of the lesion. A subsequent reconstructive surgery was also planned to reconstruct the area with bone grafts and posteriorly osseointegrated implants.

Surgical removal of the residual tumor was performed at a hospital under general anesthesia. A buccolingual mucoperiosteal flap, extending from the lower right canine to the lower right third molar, was raised, and an osteotomy was performed to separate the remaining tumor from the subjacent bone with complete curettage of the cavity (Figure 4). Histopathological examination confirmed the initial diagnosis of cementoblastoma. Microscopic examination revealed a cementum-like calcified tissue with dense trabeculae of mineralized material. The cement was intensely basophilic and exhibited irregular reversal lines (Figure 5). The presence of fibrovascular tissue cells dispersed throughout the mineralized material or at the periphery of the lesion was extensive as well as the presence of multinucleated giant cells.

In the postoperative period, the patient presented inferior alveolar nerve paresthesia, which resolved spontaneously 20 days later. Panoramic radiography performed immediately after surgery revealed complete tumor removal. The patient was monitored for 1 year after surgery, and did not show any signs of recurrence (Figure 6). After this period, he was lost upon follow-up, and the reconstruction that was originally planned was not possible.

Mandibular cementoblastoma: Case report (4)

Figure 3. Postoperative panoramic radiograph after the initial surgery showing large amounts of residual lesions and a fracture of the mesial root of the lower right third molar.

Mandibular cementoblastoma: Case report (5)

Figure 4. Residual tumor removed in the second surgery.

Mandibular cementoblastoma: Case report (6)

Figure 5. Histologic findings: Osteocementous calcified tissue with thick trabeculae of mineralized material containing numerous gaps and cementocytes. (H.E. magnification 20×).

Mandibular cementoblastoma: Case report (7)

Figure 6. Panoramic radiograph at the one year follow-up. Local bone remodeling and no signs of recurrence.

3. DISCUSSION

Cementoblastoma is a rare odontogenic neoplasm, with an estimated occurrence, according to Baart et al. [14], of less than 1 case per million people per year. Young adults who are under 30 years of age are most commonly affected [1,3,7-15,17], with a slight male prevalence [2,3,9].

According to Brannon et al. [2], 79.5% of tumors occur in the mandible, and in most cases, the lesion involves the first permanent molar [1,7,12,14,17]; in the few cases in which the upper jaw is affected, the maxillary sinus can be involved [3,13]. There are also reports of cementoblastoma associated with impacted [8,11,13] and deciduous teeth [3,10]. The occurrence of the lesion involving more than one permanent tooth, as in the case reported here, is rare [3,12-14]. Regardless of the number of teeth involved, the lesion is always associated with the dental root [1-18].

Patients may be completely asymptomatic [1,2,8]; however, bone expansion and pain can occur [2,3,7-15]. Trismus [8,10], dental displacement, and increased mobility of adjacent teeth [14] may eventually be observed.

Radiographically, the lesion presents as a well-defined radiopaque mass, surrounded by a radiolucent halo associated with the root of the tooth or teeth [1,3,7-12,14,15, 17,18]. Depending on the developmental stage of the lesion, the radiological appearance and clinical interpretation can vary, leading to an erroneous diagnosis of osteoma or osteoblastoma [8,13,14]. Other features that can eventually be observed on radiographs include root resorption [1-3,10,14,17,18], obliteration of the periodontal ligament space [2,7], and invasion of the root canals [3].

Histologically, cementoblastoma is characterized by tissue layers that are similar to cement [3,13] and, consist of poorly mineralized cellular material with prominent basophilic reversal lines [1-3,15,17,18] organized in a fibrovascular stroma [1-3,9,12,13,18]. Prominent cementoblasts [12], irregular lacunae [3,8,15], increased activity of cementoblasts and cementoclasts [2], and trabeculae of uncalcified matrix perpendicular to the surface [1,7,12,14,18] are typical characteristics of this lesion.

The most difficult challenge in the differential diagnosis of cementoblastoma is osteoblastoma. These two tumors may exhibit the same histomorphology [18], but they differ in their origin [2]. Some authors defend the odontogenic origin as a basis to differentiating cementoblastoma from osteoblastoma; this is because in cementoblastoma, the lesion is part of the root structure of the involved tooth caused by neoplastic cementoblasts that produce mineralized material that fuses with a tooth root structure [7,17,18]. Confirming this, Cundiff [7] presented a case in which the growth of a cementoblastoma was radiographically followed for more than four years. He described rare findings, such as a slight enlargement of the periodontal ligament space, until the complete removal of the tumor when it was 3 cm in diameter. Osteoblastomas are tippically separated from the adjacent tooth by a barrier that is formed by the periodontal ligament [18]. Columns of tissue that are similar to nonmineralized cementum that are located on the periphery of the mass also support the presence of a cementoblastoma [14]. Both cementoblastomas and osteoblastomas may cause root resorption [18].

In addition to osteoblastoma, the differential diagnosis of cementoblastoma must also include osteosarcoma [1], osteoma [8], focal sclerosing osteomyelitis, osteoesclerosis, and fibrous dysplasia [12,13].

The recommended treatment of cementoblastomas consists of the surgical removal of the lesion along with the tooth/teeth and/or structures that are affected, followed by complete curettage of the area or the peripheral osteotomy of the entire region [2,8,9,10,14]. When an early diagnosis is made, the treatment may involve the complete excision of the lesion with preservation of the involved tooth, thorough endodontic treatment [17] and, in some cases, apicoectomy [15]. For those cases in which a late diagnosis is made and the tumor has already achieved major proportions, as in this report, the complete removal of the lesion and associated structures is recommended due to the unlimited growth potential [8,17] and eventual recurrence. In these cases, the surgical procedure must be performed under general anesthesia [2,3,11,12,14], which ensures a less stressful intraoperative time for the patient and for the surgical team. As we observed in the present case, the initial surgery under local anesthesia resulted in surgical failure because the surgeon was not able to achieve the final objective of the complete removal of the lesion. In addition, the patient was exposed to an unnecessary psychological trauma.

Recurrences are rare if complete tumor enucleation is performed. Brannon et al. [2], stated that recurrence is more common when curettage is performed without the extraction of the involved tooth or teeth. Mandibular expansion and perforation of the cortex are clinical signs of recurrence [2,9]. Studies indicate recurrence between 6 months and 1 year after the initial surgery [2,9,14].

4. CONCLUSION

Cementoblastoma is a benign tumor with a low recurrence rate but unlimited growth potential. Appropriate treatment consists of the surgical removal, and early diagnosis favors a more conservative surgery with the possibility of preservation of the involved teeth. In cases in which the tumor is detected in advanced stages of development, the teeth should be removed along with the tumor to decrease the possibility of recurrence.

REFERENCES

  1. Hubber, A.R. and Folk, G.S. (2009) Cementoblastoma. Head and Neck Pathol, 3, 133-135. doi:10.1007/s12105-008-0099-5
  2. Brannon, R.B., Fowler, C.B., Carpenter, W.M. and Corio, R.L. (2002) Cementoblastoma: An innocuous neoplasm? A clinicopathologic study of 44 cases and review of literature with special emphasis on recurrence. Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology, and Endodontology, 93, 311-320. doi:10.1067/moe.2002.121993
  3. Ohki, K., Kumamoto, H., Nitta, Y., Nagasaka, H., Kawamura, H. and Ooya, K. (2004) Benign cementoblastoma involving multiple maxillary teeth: Report of a case with a review of the literature. Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology, and Endodontology, 97, 53-58. doi:10.1016/j.tripleo.2003.08.012
  4. Barnes, L., Eveson, J.W., Reichart, P. and Sidransky, D. (2005) World Health Organization Classification of tumours: Pathology and genetics of tumours of the head and neck. IARC Press, Lyon.
  5. Lee, Y., Xuan, M., Takata, T., Wang, C., He, Z., Zhou, Z., Mock, D. and Nickai, H. (1998) Odontogenic tumors. A demographic study of 759 cases in a chinese population. Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology, and Endodontology, 86, 707-714.
  6. Ochsenius, G., Ortega, A., Godoy, L., Penafieli C. and Escobar, E. (2002) Odontogenic Tumors in Chile: A study of 362 cases. Journal of Oral Pathology & Medicine, 31, 415-420. doi:10.1034/j.1600-0714.2002.00073.x
  7. Cundiff, E.J. (2000) Developing cementoblastoma: Case report and update of differential diagnosis. Quintessence International, 31, 191-195.
  8. Sumer, M., Gunduz, K., Sumer, P.A. and Gunhan, O. (2006) Benign cementoblastoma: A case report. Medicina Oral, Patología Oral Y Cirugía Bucal, 11, 483-485.
  9. Zaitoun, H., Kujan, O. and Sloan, P. (2007) An unusual recurrent cementoblastoma associated with a developing lower second molar tooth: A case report. Journal of Oral and Maxillofacial Surgery, 65, 2080-2082. doi:10.1016/j.joms.2006.06.288
  10. Vieira, A.P.G.F., Meneses, J.M.S. Jr. and Maia, R.L. (2007) Cementoblastoma related to a primary tooth: A case report. Journal of Oral Pathology & Medicine, 36, 117-119. doi:10.1111/j.1600-0714.2007.00465.x
  11. Piattelli, A., Di Alberti, L., Scarano, A. and Piatelli, M. (1998) Benign cementoblastoma associated with an unerupted third molar. Oral Oncology, 34, 229-231.
  12. Pontes, F.S.C., Carneiro, J.T. Jr., Ribeiro, A.L.R., Gonçalves, A. Jr., Fonseca, F.P., Pontes, H.A.R. and Pinto, D.S. Jr. (2008) Cementoblastoma previously misdiagnosed as fibrous dysplasia: Report of an uncommon case and discussion of the differential diagnosis. International Journal of Pediatric Otorhinolaryngology Extra, 3, 182-187. doi:10.1016/j.pedex.2008.04.001
  13. Infante-Cossio, P., Hernandez-Guisado, J.M., Acosta-Feria, M. and Carranza-Carranza, A. (2008) Cementoblastoma involveing the maxillary sinus. British Journal of Oral and Maxillofacial Surgery, 46, 234-236. doi:10.1016/j.bjoms.2007.03.009
  14. Baart, J.A., Lekkas, C. and Van der Waal, I. (1991) Residual cementoblastoma of the mandible. Journal of Oral Pathology & Medicine, 20, 300-302. doi:10.1111/j.1600-0714.1991.tb00932.x
  15. Hirai, E., Yamamoto, K., Kounoe, T., Kondo, Y., Yonemasu, H. and Kurokawa, H. (2010) Benign cementoblastoma of the anterior maxilla. Journal of Oral and Maxillofacial Surgery, 68, 671-674. doi:10.1016/j.joms.2009.03.060
  16. Ulmanski, M., Hansen, E., Praetorius, F. and Haque, M.F. (1994) Benign Cementoblastoma: A review and five new cases. Oral Surgery, Oral Medicine, Oral Pathology, 77, 48-55. doi:10.1016/S0030-4220(06)80106-4
  17. Biggs, J.T. and Benenati, F.W. (1995) Surgically treating a benign cementoblastoma while retaining the involved tooth. Journal of the American Dental Association, 126, 1288-1290.
  18. Slootweg, P.J. (1992) Cementoblastoma and osteoblastoma: A comparison of histologic features. Journal of Oral Pathology & Medicine, 21, 385-389. doi:10.1111/j.1600-0714.1992.tb01024.x

NOTES

*Corresponding author.

Mandibular cementoblastoma: Case report (2024)

FAQs

What is the difference between cementoma and cementoblastoma? ›

Cementomas develop as cells that generate cementum, or cementoblasts uncontrollably proliferate at the apex of a tooth root. Cementoblasts that form the cementum typically cease activity and become cementocytes.

Is cementoblastoma benign or malignant? ›

True cementoma, now known as cementoblastoma, is a benign odontogenic tumor commonly presenting with painful swelling of the alveolar ridges (1). Such lesions have a predilection for the mandible and are primarily associated with the mandibular first molar.

Does cementoblastoma need to be removed? ›

The cementoblastoma has been described as a benign, solitary, slow-growing lesion, although there have been reports of aggressive behaviour. Due to the benign neo plastic nature of the lesion, the treatment of choice is complete removal of the lesion with extraction of the associated tooth.

What is the treatment for cementoblastoma? ›

The most common therapy for cementoblastoma is complete enucleation associated with extraction of the involved tooth (3,4,9). Tooth extraction is recommended to simplify the differential diagnosis between cementoblastoma and other lesions not involving the dental root during histological examination (26).

Does Cementoblastoma cause root resorption? ›

On x ray, characteristic findings of cementoblastoma include well defined, markedly radiopaque mass, radiolucent peripheral "line" which overlies and obliterates the tooth root, apparent external root resorption, and severe hypercementosis.

Why is Cementoblastoma painful? ›

Cementoblastoma has, only rarely, been associated with a primary or impacted tooth [1–4]. All cases are connected to the root of the involved tooth [1–7]. Cementoblastoma commonly presents with pain and associated swelling due to bony expansion of the buccal and lingual aspects of the alveolar ridges [1–5].

Is tooth vital in cementoblastoma? ›

But may be associated with diffuse pain and tooth mobility, but the tooth is still vital. Since a cementoblastoma is a benign neoplasm, it grossly forms a mass of cementum-like tissue as an irregular or round mass attached to the roots of a tooth, usually the permanent mandibular first molar.

How long does osteoid osteoma last? ›

NSAIDs help to relieve pain and may speed shrinkage of the osteoid osteoma. With this conservative treatment, symptoms usually end within 33 months (2.75 years). Providers may suggest surgery to treat an osteoid osteoma.

Can cementoma cause root resorption? ›

The most commonly observed features of benign cementoblastoma are the expansion of cortical plates and root resorp- tion of the affected tooth because benign cementoblastoma is probably truly neoplastic in contrast to PCD 3,~9,2~ Our cases showed the expansion of cortical plates and root resorption.

Where are cementoblasts located? ›

A cementoblast is a biological cell that forms from the follicular cells around the root of a tooth, and whose biological function is cementogenesis, which is the formation of cementum (hard tissue that covers the tooth root).

What is a traumatic bone cyst? ›

Traumatic bone cyst, or simple bone cyst, is a pseudocyst that represents a bony dead space. Although trauma is suspected to be the cause, this has not been proved. The lesion is believed to develop from abnormal healing of hemorrhage within bone.

How is ameloblastoma treated? ›

Ameloblastoma treatment usually includes surgery to remove the tumor. Ameloblastoma often grows into the nearby jawbone, so surgeons may need to remove the affected part of the jawbone. An aggressive approach to surgery reduces the risk that ameloblastoma will come back. Surgery to repair the jaw.

What is Pindborg tumor? ›

Calcifying epithelial odontogenic tumor, also known as Pindborg tumor, is a rare, benign, locally aggressive tumor that occurs in the same age range and in the same jaw sites as ameloblastoma. There is no gender predilection. A propensity for the mandibular molar region versus the premolar maxilla has been reported.

What is an Odontome? ›

What is an Odontome? Odontomata are benign calcifying hamartomata and are the most common type of odontogenic harmartoma among patients younger than 20 years. These hamartomata are composed of various dental tissues, that is, enamel, dentine, cementum and sometimes pulp.

What is periapical Cemental dysplasia? ›

Periapical cemento-osseous dysplasia (PCOD) is a rare benign lesion, often asymptomatic, in which fibrous tissue replaces the normal bone tissue, with metaplasic bone and neo-formed cement.

What causes bulbous tooth roots? ›

Hypercementosis is an idiopathic, non-neoplastic condition characterized by the excessive buildup of normal cementum (calcified tissue) on the roots of one or more teeth. A thicker layer of cementum can give the tooth an enlarged appearance, which mainly occurs at the apex or apices of the tooth.

How do you treat condensing osteitis? ›

How is condensing osteitis treated?
  1. a root canal to help treat inflammation and any infections around the affected tooth.
  2. antibiotics for any existing infections.
  3. extraction for the affected tooth if permanent damage to the pulp is suspected.
16 Jun 2021

What causes cementoma? ›

The cause of the cementoma continues to be unknown and is linked with many sources, including trauma, nutritional deficiency, metabolic disturbances, constitutional factors, and others. Zegarelli and Kutscherl' have collected data which suggest an association with an endocrine disturbance of an unknown nature.

What are the two types of cementum? ›

There are two types of cementum: acellular and cellular. Both consist of a calcified interfibrillar matrix and collagen fibrils. The cellular type contains cementocytes in individual spaces (lacunae) that allow for communication with each other through a system of canaliculi. There are two types of collagen fibers.

What is idiopathic sclerosis? ›

Idiopathic osteosclerosis (IO) is an area of increased bone production in the jaw and generally appears to be round, elliptical, or irregular and radiopaque in shape.

What is an odontogenic tumor? ›

Odontogenic tumors are any kind of abnormal growth in and around the jaw and teeth; many of these tumors are considered to be benign. In unusual cases, odontogenic tumors are malignant, meaning they are likely to spread.

What causes periapical Cemento osseous dysplasia? ›

Periapical cemento-osseous dysplasia (COD) is a very rare benign lesion arising from a group of disorders which are known to originate from undifferentiated cells of the periodontal ligament tissue. Essentially, these underlying disorders all involve the same pathological process.

What is a Dentigerous cyst? ›

Abstract. Dentigerous cyst is a type of odontogenic cysts and generally occurs in the ages of twenties or thirties. Dentigerous cyst always includes a tooth which cannot complete the eruption process and occurs around the crown by the fluid accumulation between the layers of enamel organ.

What is sclerosing osteitis? ›

Sclerosing osteomyelitis (condensing osteitis, bony scar, sclerotic bone, focal periapical osteopetrosis) is an inflammatory condition believed to be a local bony reaction to a low-grade inflammatory stimulus or to bacteria of low virulence.

Can osteoma cancerous? ›

Osteomas are benign head tumors made of bone. They're usually found in the head or skull, but they can also be found in the neck. While osteomas are not cancerous, they can sometimes cause headaches, sinus infections, hearing issues or vision problems – however, many benign osteomas don't require treatment at all.

Is it necessary to remove osteoma? ›

In the absence of symptoms, there is no need to remove osteomas by surgery. However, it is necessary to remove osteomas to preserve major organs and for aesthetic purposes.

How is an osteoma removed? ›

Using a classic approach, forehead osteoma removal may be performed by making a small buttonhole incision just inside the hairline or scalp. The bone is then reshaped using an endoscopic procedure, where excess osteocytes (bone cells) are removed.

What is Ameloblastic fibroma? ›

Ameloblastic fibroma (AF) is an extremely rare true mixed benign tumor that can occur either in the mandible or maxilla.[1] It is frequently found in the posterior region of the mandible, often associated with an unerupted tooth.[2] It usually occurs in the first two decades of life with a slight female predilection, ...

What is odontogenic Keratocyst? ›

Introduction. Odontogenic keratocysts (OKCs), first described by Philipsen in 1956 [1], are benign intraosseous lesions of odontogenic origin that account for about 10% of jaw cysts. They are characterised by an aggressive behaviour with a relatively high recurrence rate [2].

What is Adenomatoid odontogenic tumor? ›

Adenomatoid odontogenic tumor (AOT) is a rare tumor of epithelial origin comprising 3% of all the odontogenic tumors. It is a benign, painless, noninvasive, and slow-growing lesion, with a relative frequency of 2.2-13% and often misdiagnosed as an odontogenic cyst on clinical examination.

What is the origin of cementoblasts? ›

Abstract. It is generally accepted that cementoblasts originate in the process of differentiation of the mesenchymal cells of the dental follicle.

What gives rise to cementoblasts? ›

Cementum formation (Cementogenesis): After completion of the root, fragmentation of the Hertwig's root sheath occurs. This process enables the cells from the adjacent dental follicle to come into contact with the root dentine. These cells further differentiate to form cementum-forming cells termed as cementoblasts.

What is Sharpey's fibers? ›

Sharpey's fibres (bone fibres, or perforating fibres) are a matrix of connective tissue consisting of bundles of strong predominantly type I collagen fibres connecting periosteum to bone.

Do bone cysts need to be removed? ›

Bone cysts are not always treated

A bone cyst may not need treatment if it's small and not causing any problems. They often get better on their own, especially in children and teenagers. You may have regular X-rays for a few years to check your bone is healing and the cyst is not getting bigger.

How long does it take for a bone cyst to heal? ›

The cyst will likely be reinjected at regular intervals every several months, until there is adequate healing, which may take six to 12 months.

How are bone cysts treated? ›

Bone Cyst Treatment

Most often bone cysts resolve with time. Repeated X-rays are usually done to make sure the cyst is getting smaller. If a pathologic fracture occurs, the cyst may need to be surgically cleaned and then packed with bone graft to fill in the bone and encourage bone healing.

Can ameloblastoma cause death? ›

In general, ameloblastoma is not fatal as it is a benign condition. If ameloblastoma occurs in the maxillary region, then it can be a fatal condition due to the thinning of the bones. In some patients, the tumor can spread to the sinonasal passages that can affect the brain.

Can ameloblastoma become cancerous? ›

Rarely, ameloblastoma can become cancerous (malignant). Very rarely, ameloblastoma cells can spread to other areas of the body (metastasize), such as the lymph nodes in the neck and lungs. Ameloblastoma may recur after treatment.

How fast does ameloblastoma grow? ›

Ameloblastomas grow very slowly. You can have an ameloblastoma for 10 to 20 years before you or your healthcare provider notice potential problems. Ameloblastomas can change the shape of your face and jaw or cause problems with your teeth.

Is hamartoma benign or malignant? ›

A benign (not cancer) growth made up of an abnormal mixture of cells and tissues normally found in the area of the body where the growth occurs.

What is another name for ameloblastoma? ›

It was recognized in 1827 by Cusack. This type of odontogenic neoplasm was designated as an adamantinoma in 1885 by the French physician Louis-Charles Malassez. It was finally renamed to the modern name ameloblastoma in 1930 by Ivey and Churchill.

Is OKC a cyst or tumor? ›

The odontogenic keratocyst (OKC) is an enigmatic developmental cyst that deserves special attention. It has characteristic histopathological and clinical features; but, what makes this cyst special is its aggressive behavior and high recurrence rate.

Is odontome and odontoma same? ›

Odontome in medicine and dentistry was originally used for any tumor and/or tumor-like lesion, like neoplastic cyst arising from tooth forming tissues. Odontomas are hamartomas of aborted tooth formation which account for 22% of the odontogenic tumors.

Which is most frequently affected by root Dilaceration? ›

Results: Root dilaceration was detected in 0.3% of teeth and 7.2% of patients. It was distributed equally between the maxilla and mandible. Mandibular second molar was the most frequent dilacerated tooth (1.6%) followed by maxillary first molar (1.3%) and mandibular first molar (0.6%).

Should odontomas be removed? ›

While an odontoma is a tumor, it's a benign one and not uncommon. That alone is great news! However, odontomas usually require surgical removal. They're made up of dental tissue that resembles abnormal teeth or calcified mass that invade the jaw around your teeth and could affect how your teeth develop.

Is Cementoblastoma cancerous? ›

Cementoblastoma is a type of benign odontogenic tumor, with the optimal treatment consisting of tooth extraction and follow-up examination. Osteosarcoma of the jaw is a rare, malignant disease with a poor prognosis, and the imaging and clinical appearance of the lesion is highly variable.

What is the treatment of periapical Cemental dysplasia? ›

No treatment is required, as it is harmless, and only periodic observation is required. It is difficult to radiographically distinguish this lesion from a periapical granuloma. Hence to make this distinction, vitality testing of the pulp should be done. No treatment is required for this condition.

How common is periapical Cemental dysplasia? ›

A novel finding was the occurrence of cemental dysplasia affecting the periapical area of vital mandibular teeth in 8 adult women with NF1. Thus, cemental dysplasia was detected in 34.8% of the adult female NF1 patients, while cemental dysplasia was not present in men or children with NF1.

What is a cementoma? ›

Cementomas are benign jaw tumors that originate from periodontal ligament elements. Four classifications of cementomas are described: periapical fibrous dysplasia, benign cementoblastoma, cementifying fibroma, and florid osseous dysplasia.

What are the two types of cementum? ›

There are two types of cementum: acellular and cellular. Both consist of a calcified interfibrillar matrix and collagen fibrils. The cellular type contains cementocytes in individual spaces (lacunae) that allow for communication with each other through a system of canaliculi. There are two types of collagen fibers.

What causes a cementoma? ›

The cause of the cementoma continues to be unknown and is linked with many sources, including trauma, nutritional deficiency, metabolic disturbances, constitutional factors, and others. Zegarelli and Kutscherl' have collected data which suggest an association with an endocrine disturbance of an unknown nature.

What is an Odontome? ›

What is an Odontome? Odontomata are benign calcifying hamartomata and are the most common type of odontogenic harmartoma among patients younger than 20 years. These hamartomata are composed of various dental tissues, that is, enamel, dentine, cementum and sometimes pulp.

What is Pindborg tumor? ›

Calcifying epithelial odontogenic tumor, also known as Pindborg tumor, is a rare, benign, locally aggressive tumor that occurs in the same age range and in the same jaw sites as ameloblastoma. There is no gender predilection. A propensity for the mandibular molar region versus the premolar maxilla has been reported.

What is Cemental dysplasia? ›

Abstract. Focal cemento-osseous dysplasia (FCOD) is a benign fibro-osseous lesion of bone characterized by the replacement of normal bone by fibrous tissue and subsequently followed by its calcification with osseous and cementum-like material. It is mostly asymptomatic in nature and requires no treatment.

What is a traumatic bone cyst? ›

Traumatic bone cyst, or simple bone cyst, is a pseudocyst that represents a bony dead space. Although trauma is suspected to be the cause, this has not been proved. The lesion is believed to develop from abnormal healing of hemorrhage within bone.

What type of collagen is in PDL? ›

Like other ligaments, PDL is composed predominantly of type I collagen. Collagen synthesis is a complex process with multiple steps and numerous post-translational modifications including hydroxylation, glycosylation, and cross-linking.

What is the other name of cementum? ›

Cementum, or root cementum, is a mineralized tissue covering the entire root surface.

Where is cementum thickest? ›

Structure of cementum

The thickness of the cementum varies at different levels of the tooth root. It is thickest at the apex of the root and between two roots when a tooth has more than one root. It is thinnest at the upper part of the root where it is 10-15µm thick.

What causes periapical Cemental dysplasia? ›

Periapical cemento-osseous dysplasia (COD) is a very rare benign lesion arising from a group of disorders which are known to originate from undifferentiated cells of the periodontal ligament tissue. Essentially, these underlying disorders all involve the same pathological process.

What causes Cemento-osseous dysplasia? ›

Cemento-osseous dysplasia (COD) is a benign condition of the jaws that may arise from the fibroblasts of the periodontal ligaments.
...
Cemento-osseous dysplasia
ComplicationsInfection with extractions or abscessed teeth, unnecessary root canals
Usual onset20-30 years
TypesLocalized, generalized
CausesCongenital
8 more rows

What causes Hypercementosis? ›

There are reports of familial cases making hypercementosis an inheritable condition. The local factors implicated to cause hypercementosis are occlusal trauma,7 inflammation secondary to pulpal or periodontal disease, tooth mobility,2 repair of root fracture and transplantation of teeth.

Is odontome and odontoma same? ›

Odontome in medicine and dentistry was originally used for any tumor and/or tumor-like lesion, like neoplastic cyst arising from tooth forming tissues. Odontomas are hamartomas of aborted tooth formation which account for 22% of the odontogenic tumors.

Are odontomas cancerous? ›

Odontomas are not cancer. They are considered benign tumors, though in humans they are often surgically removed.

Which is most frequently affected by root Dilaceration? ›

Results: Root dilaceration was detected in 0.3% of teeth and 7.2% of patients. It was distributed equally between the maxilla and mandible. Mandibular second molar was the most frequent dilacerated tooth (1.6%) followed by maxillary first molar (1.3%) and mandibular first molar (0.6%).

Top Articles
Latest Posts
Article information

Author: Cheryll Lueilwitz

Last Updated:

Views: 6317

Rating: 4.3 / 5 (74 voted)

Reviews: 81% of readers found this page helpful

Author information

Name: Cheryll Lueilwitz

Birthday: 1997-12-23

Address: 4653 O'Kon Hill, Lake Juanstad, AR 65469

Phone: +494124489301

Job: Marketing Representative

Hobby: Reading, Ice skating, Foraging, BASE jumping, Hiking, Skateboarding, Kayaking

Introduction: My name is Cheryll Lueilwitz, I am a sparkling, clean, super, lucky, joyous, outstanding, lucky person who loves writing and wants to share my knowledge and understanding with you.